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Medical AACN-CMC : Cardiac Medicine Subspecialty Certification Exam

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Test Number : AACN-CMC
Test Name : Cardiac Medicine Subspecialty Certification
Vendor Name : Medical
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AACN-CMC exam Format | AACN-CMC Course Contents | AACN-CMC Course Outline | AACN-CMC exam Syllabus | AACN-CMC exam Objectives


CMC® is a subspecialty certification for certified nurses who provide direct care to acutely/critically ill adult cardiac patients. Nurses interested in this certification may work in areas such as cardiac care units, medical ICUs, telemetry, progressive care, heart failure clinics, home care, interventional cardiology, cardiac cath labs and/or electrophysiology units.

A current, unencumbered U.S. RN or APRN license is required.
An unencumbered license is not currently being subjected to formal discipline by any state board of nursing and has no provisions or conditions that limit the nurses practice in any way.

A current nationally accredited clinical nursing specialty certification, to which the CMC credential will be attached, is required.

Candidates must complete one of the following clinical practice hour requirement options:

Two-Year Option

Practice as an RN or APRN for 1,750 hours in direct care of acutely/critically ill adult patients during the previous two years, with 875 of those hours accrued in the most accurate year preceding application. Of those 1,750 hours, 875 need to be in the care of acutely/critically ill adult cardiac patients.
OR
Five-Year Option
Practice as an RN or APRN for at least five years with a minimum of 2,000 hours in direct care of acutely/critically ill adult patients, with 144 of those hours accrued in the most accurate year preceding application. Of those 2,000 hours, 1,000 need to be in the care of acutely/critically ill adult cardiac patients.

Must be completed in a U.S.-based or Canada-based facility or in a facility determined to be comparable to the U.S. standard of acute/critical care nursing practice.
Are those spent actively providing direct care to acutely/critically ill adult patients or supervising nurses or nursing students at the bedside of acutely/critically ill adult patients, if working as a manager, educator, preceptor or APRN.
Are verifiable by your clinical supervisor or professional colleague (RN or physician). Contact information must be provided for verification of eligibility related to clinical hours, to be used if you are selected for audit.
For complete eligibility requirements.



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Medical Cardiac Study Guide

examine reveals cardiac protein as a brand new target for various kinds of heart ailment | AACN-CMC exam Questions and PDF Dumps

Like a failing gasoline pump that causes a lack of engine power in a car, a diseased coronary heart can take a significant toll on the physique's performance. For some sufferers, projects like running up a flight of stairs or walking across a room eventually become laborious endeavors. here's as a result of, over time, inspite of the underlying trigger, heart hurt typically progresses, because of a continuing barrage of oxidative stress and poisonous lipids that alter coronary heart cell energetics and, ultimately, the skill of the heart to function constantly.

Oxidative stress happens when hazardous oxygen-containing molecules outnumber valuable antioxidants, resulting in damaging reactions with proteins, DNA, and different cellphone add-ons. Now, in two new reports, researchers on the Lewis Katz faculty of drugs at Temple institution (LKSOM) exhibit that in the coronary heart, one molecule in specific, Kruppel-like aspect (KLF)-5, single-handedly fuels both the technology of oxidizing molecules and the accumulation of poisonous lipids known as ceramides within the heart, exacerbating heart dysfunction. The stories are the first to establish KLF5 as a typical mediator of cardiac harm in animal models of distinct diseases that lead to abnormal coronary heart function, including diabetes and coronary heart assault.

Our findings expose KLF5 as a new target for several types of cardiac disease. As a unifying aspect riding oxidative stress and accumulation of poisonous lipids within the heart, the implications of targeting KLF5 may be a long way-accomplishing, opening up remedy for a broad range of illnesses involving heart dysfunction."

Konstantinos Drosatos, PhD, associate Professor of Pharmacology at the core for Translational medicine, the middle for Metabolic disorder research, and Alzheimer's center at Temple at LKSOM

within the first analyze, posted on-line December 2 within the journal Circulation analysis, Dr. Drosatos and colleagues investigated the involvement of KLF5 in a mouse mannequin of diabetic cardiomyopathy. Diabetic cardiomyopathy is an enormous complication of diabetes and is characterized in selected by altered heart mobilephone metabolism and oxidative hurt. the new look at confirmed that sufferers with diabetes have excessive levels of KLF5 expression in the heart.

The researchers found that mice with diabetic cardiomyopathy in a similar way have high KLF5 expression, and that they discovered that increased KLF5 is linked to the build up of ceramides in the coronary heart. Ceramides, which ensue naturally within the cellphone membrane, are time-honored to reach toxic ranges in the presence of insulin resistance and extreme heart harm, corresponding to that inflicted via heart attack.

The Temple group confirmed that in mice, however, these dangerous outcomes may be halted. "Inhibiting KLF5 with a drug, in addition to with genetic interventions, now not only reduced oxidative stress and avoided ceramide accumulation but additionally restored cardiac feature," Dr. Drosatos defined.

within the 2d analyze, published on-line January 12 within the journal Circulation, which turned into pushed through LKSOM MD-PhD scholar, Matthew Hoffman, Dr. Drosatos's group investigated the function of KLF5 in mice with heart failure prompted with the aid of cardiac ischemia, a surprising, extreme blockage of blood move to the heart. Cardiac ischemia in general is adopted by means of extensive raises in poisonous lipids, especially ceramides. The researchers had been able to display that KLF5 is panic in inflicting the construction of ceramides that underlies damage to the cardiac wall. Ceramide buildup changed into driven by KLF5-induced overexpression of a molecule called SPT1.

"The next step is to assess even if the severity of heart disease or the manner sufferers respond to medication is linked to increased KLF5," Dr. Drosatos said. "We be aware of from clinical observations, for example, that some patients with heart failure are less responsive than others to therapeutic interventions. They wish to understand no matter if KLF5 is an element that defines how well the sufferers will respond to treatments."

moreover, Dr. Drosatos and colleagues plan to search for proteins that regulate KLF5, which could develop realizing of the position and pathway of activation of KLF5 in the heart and different tissues.

The studies additionally mark new ground in individual and collaborative research efforts at Temple college.


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